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Intriguingly, both the platypus and echidna GLP-1 peptides displayed a distinct pattern of signalling in comparison to hGLP-1 and the clinically approved mimetic Ex-4, which was apparent at both the human and platypus GLP-1 receptors (Fig

Direct binding of copper ions to fatty acylated components in the tricarboxylic acid (TCA) cycle results in protein aggregation and dysregulation (40), blocking the TCA cycle and inducing proteotoxic stress and cell death (Fig

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The absence of drug affinity complex modification results in a shorter receptor interaction window, allowing for transient activation that aligns with pulsatile signaling models

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