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(viii) improve mitochondrial dysfunction that has been associated with IR and (ix) achieve -cell function preservation (both in patients with T2D and individuals with prediabetes) and suppress excess intra-pancreatic fat deposition [44, 45, 5764] (Figure 1)

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With branded supply now stable, the FDA determined there is no longer a clinical need for outsourcing facilities to compound these medications from bulk drug substances

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