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A new preclinical study at Northwestern Universitys Feinberg School of Medicine found that disrupting complex I function in mouse dopaminergic neurons led to progressive PD-related motor deficits, with loss of dopamine release in the substantia nigra being critical for motor deficits (40)

These observations seem to be associated with the inhibition of Na + /H + exchanger 3 (NHE3) in the proximal tubules

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