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Meanwhile, InsR levels were unaffected by mAb treatment in all tissues examined for either sex (Fig

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104 In GBM cells, histone H3 lactylation, particularly H3K14/18la, facilitates Wnt/-catenin, NF-B, and PD-L1 expression, thereby enhancing CD8 + T-cell infiltration and contributing to immune evasion in GBM

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