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D., Valentini, G., Bianchi, G., Biasi, G., Cacace, E., Adami, S
At the systemic level, GLP-1 RAs improve insulin sensitivity in peripheral tissues (muscle, adipose), reduce circulating free fatty acid flux to the liver, suppress hypothalamic appetite circuits to promote weight loss, and lower systemic pro-inflammatory cytokines (TNF-, IL-1, IL-6), thereby reducing the hepatic inflammatory milieu indirectly [7,26,30]
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GLP-1 drugs are not just gut hormones, they cross the blood-brain barrier and bind to receptors in the mesolimbic system, which modulates reward signalling
The next frontier of GLP-1 competition may lie not only in the molecule itself, but in how it is delivered
Comparative analysis of different peptidyl-prolyl isomerases reveals FK506-binding protein 12 as the most potent enhancer of alpha-synuclein aggregation
