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GLP-1 Receptor Agonism Mechanism Stimulates glucose-dependent insulin release from pancreatic -cells, reducing hyperglycaemia
Investig Clin Urol 58(Suppl 2):S90-S98
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SMAD3 represses insulin gene transcription and impairs -cell function, while its deficiency improves glucose tolerance, insulin sensitivity, and diabetic kidney injuries (Lin et al
This suggests that a resistance exercise-related mechanism of inducing plasma NO, perhaps due to increased shear stress that triggered an up-regulation in NO-cGMP signaling along with a slow, sustained release of NO from GSH + CIT, may be a conceivable candidate for this response
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