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This strategy has been applied to dulaglutide, where two GLP-1 molecules are fused to the Fc fragment of IgG4, increasing the half-life while reducing the renal elimination due to the increased molecular weight
Notably, AMPK inhibition abolishes these benefits, confirming that -PAEs protective effects are AMPK-dependent (50)
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loose stool which resolved upon dose reduction (unknown grading
Whilst GLP-1 receptor agonists are not licensed for treating inflammatory diseases, emerging clinical evidence suggests potential benefits in several conditions characterised by chronic inflammation
Ferrostatin-1 reduces ferroptotic vulnerability suggesting an important role for lipid peroxidation in this process
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