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Potential off-target effects, such as promoting cancer, serve as a crucial reminder that even protective pathways have context-dependent effects, echoing the delicate balance of redox signaling versus distress laid out in Section 2.2
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CrgA, a LysR-type regulator that is upregulated upon contact with human epithelial cells and represses its own transcription and that of the type IV pili subunits pilE and pilC1
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