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Unlike the kinases for which multiple inhibitors have been developed and tested in clinical trials, the most common palmitoylation inhibitor, 2-BP, cannot be used clinically because of its significant off-target activity and toxicity, including inhibition of mitochondrial fatty acid oxidation (247, 248)
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Mechanistically, GHRP-6 sustained cellular antioxidant defense, upregulated the prosurvival gene Bcl-2, and preserved cardiomyocyte mitochondrial integrity
Colleagues he consulted hadnt heard of it either
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