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Its important to check how well the treatment is working and adjust it if needed
The tolerability gap may be less significant in practice than the raw nausea percentages suggest
In multiple sclerosis models, GLP-1 receptor activation reduces pathogenic Th1 and Th17 cell activity, inhibits microglial activation (the brain equivalent of macrophage inflammation), and may enhance remyelination processes
Individual tolerability varies
They reduce de novo lipogenesis by inhibiting transcription factor sterol regulatory element-binding protein 1c (SREBP-1c), which is a master regulator of fatty acid synthesis (Shantaram et al., 2025
Wilding JPH, et al
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