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Type IV pili binding to CD147/Basigin triggers tyrosine phosphorylation through c-Src and focal adhesion kinase, and EGFR is activated (Slanina et al., 2010, Slanina et al., 2012, Slanina et al., 2014), and acid sphingomyelinase increases ceramide levels on the plasma membrane, forming platforms for the recruitment of host receptors (Simonis et al., 2014

Cullin, N., Azevedo Antunes, C., Straussman, R., Stein-Thoeringer, C

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To determine if the same pattern of biomarkers of abnormal fatty-acid metabolism are present in children with ASD, the laboratory results from a large cohort of children with ASD ( n =213) who underwent screening for metabolic disorders, including mitochondrial and fatty-acid oxidation disorders, in a medically based autism clinic were reviewed

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